Calcitriol (INN) l, also called 1,25-dihydroxycholecalciferol or 1,25-dihydroxyvitamin D3, is the hormonally active form of vitamin D with three hydroxyl groups (abbreviated 1,25-(OH)2D3 or simply 1,25(OH)2D), It was first identified by Michael F. Holick in work published in 1971. Calcitriol increases the level of calcium (Ca2+) in the blood by in...
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http://en.wikipedia.org/wiki/Calcitriol
The active form of vitamin D. Calcitriol is formed in the kidneys or made in the laboratory. It is used as a drug to increase calcium levels in the body in order to treat skeletal and tissue-related calcium deficiencies caused by kidney or thyroid disorders.
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http://www.cancer.gov/dictionary?expand=C
1_, 25 dihydroxyvitamin D3. The form of vitamin D3 that is biologically active in intestinal transport and calcium resorption by bone. ... (18 Nov 1997) ...
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http://www.encyclo.co.uk/local/20973
(kal″sĭ-tri´ol) 1,25-dihydroxycholecalciferol; a form of vitamin D used as a calcium regulator in the management of hypocalcemia in conditions such as rickets, osteodystrophy, hypoparathyroidism, and complications of renal dialysis; administered orally or intravenously.
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http://www.encyclo.co.uk/local/21001
(= 1a, 25-dihydroxyvitamin D3.) The form of vitamin D3 that is biologically active in intestinal transport and calcium resorption by bone.
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Type: Term Pronunciation: kal′si-trī′ol Definitions: 1. formation of calcitriol is the second step in the biologic conversion of vitamin D
3 to its active form; it is more potent than calcidiol.
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http://www.medilexicon.com/medicaldictionary.php?t=13301
Calcitriol: The active form of vitamin D. Calcitriol is formed in the kidneys or made in the laboratory. It is used as a drug to increase calcium levels in the body in order to treat skeletal and tissue-related calcium deficiencies caused by kidney or thyroid disorders.
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http://www.medterms.com/script/main/art.asp?articlekey=21111
The active form of 1, 25 dihydroxycholecalciferol (vitamin D). Can be exogenously administered to patients with chronic renal failure to suppress PTH secretion by the parathyroid glands with reversal of some of the clinical manifestations and possibly a resultant slowing of the rate of progression of renal dysfunction.
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